NIH Deep Phenotyping and Multimodal Studies
1 Walitt et al. 2024 — Deep Phenotyping Study
Full Citation:: Walitt B, Singh K, LaMunion SR, et al. Deep phenotyping of post-infectious myalgic encephalomyelitis/chronic fatigue syndrome. Nature Communications. 2024;15(1):907. (Walitt et al. 2024) DOI:: 10.1038/s41467-024-45107-3 Key Findings::
- Altered effort preference rather than physical or central fatigue
- CSF catechol abnormalities; B-cell abnormalities; sex-specific gene expression
- Reduced peak VO~2~ and chronotropic incompetence on CPET
Relevance to Part V:: Foundational multimodal dataset for integrated systems models; provides simultaneous immune, metabolic, and neurological parameters.
2 Heng et al. 2025 — Mapping the Complexity of ME/CFS
Full Citation:: Heng RB, Gunasegaran B, Krishnamurthy S, et al. Mapping the complexity of ME/CFS: Evidence for abnormal energy metabolism, altered immune profile, and vascular dysfunction. Cell Reports Medicine. 2025;6(12):102514. (Heng et al. 2025) DOI:: 10.1016/j.xcrm.2025.102514 Key Findings::
- Multi-omics evidence for abnormal energy metabolism, altered immune profile, and vascular dysfunction
- Integrates multiple pathophysiological domains in a single cohort
Relevance to Part V:: Key data source for integrated systems models linking energy, immune, and vascular subsystems.
3 DecodeME 2025 — ME/CFS Genome-Wide Association Study
Full Citation:: DecodeME Consortium, Ponting CP, et al. Initial Findings from the DecodeME Genome-Wide Association Study of Myalgic Encephalomyelitis/Chronic Fatigue Syndrome. medRxiv. 2025. (DecodeME Consortium, Ponting, et al. 2025) DOI:: 10.1101/2025.01.13.25320567 Key Findings::
- Largest ME/CFS GWAS to date ($n$=21,620)
- Preprint; identifies genetic risk loci for ME/CFS
Relevance to Part V:: Genetic architecture data for modeling genetic susceptibility and gene–environment interactions.