Neurological and Neuroendocrine Dysfunction
1 Dantzer 2007 — Cytokine-Induced Sickness Behaviour
[/ Full Citation:: Dantzer R. Twenty years of research on cytokine-induced sickness behavior. Neuroscience & Biobehavioral Reviews. 2008;32(1):220–224. (Dantzer 2008)], DOI:: 10.1016/j.neubiorev.2007.07.014 Key Findings::
- Reviews two decades of research on how peripheral cytokines induce CNS sickness behavior
- Establishes the neuroimmune basis for fatigue, anhedonia, and social withdrawal
Relevance to Part V:: Foundational framework for neuroendocrine models linking peripheral inflammation to central symptoms.
2 Raijmakers et al. 2021 — Neuroinflammation and Microglia
Full Citation:: Raijmakers R, Teunissen S, Moens B, Daemen MA, Adriaensen D. Neuroinflammation and microglia in chronic pain and itch. Frontiers in Pain Research. 2021;2:78. (Raijmakers et al. 2021) DOI:: 10.3389/fpain.2021.644542 Key Findings::
- Reviews microglial activation and neuroinflammatory pathways in chronic pain
- Links peripheral immune signals to central sensitization
Relevance to Part V:: Microglial activation dynamics for neuroinflammation sub-models in neuroendocrine modeling.
3 Novak et al. 2022 — Cerebral Blood Flow Regulation Review
Full Citation:: Novak V, Haight JL, Cooper G, Novak CM. Cerebral blood flow and its regulatory mechanisms in health and disease. Comprehensive Physiology. 2022;12(3):3125–3176. (Novak et al. 2022) DOI:: 10.1002/cphy.c210011 Key Findings::
- Comprehensive review of cerebral blood flow regulatory mechanisms
- Covers autoregulation, neurovascular coupling, and disease-related impairments
Relevance to Part V:: Reference physiology for cerebral blood flow sub-models in cardiovascular and neuroendocrine chapters.
4 Marty et al. 2008 — Nucleus Tractus Solitarius
Full Citation:: Marty S, Thorne R, Ansonoff M, et al. Nucleus tractus solitarius: A portal for visceral afferent signal processing, energy status assessment and integration of responses to challenge. Frontiers in Neuroendocrinology. 2008;29(1):27–54. (Marty et al. 2008) DOI:: 10.1016/j.yfrne.2007.07.001 Key Findings::
- NTS as central hub for visceral afferent integration and energy status monitoring
- Links autonomic, endocrine, and metabolic signaling pathways
Relevance to Part V:: Neuroanatomical basis for neuroendocrine integration models connecting autonomic dysfunction to energy sensing.
5 Cleare et al. 1999 — Low-Dose Hydrocortisone RCT
Full Citation:: Cleare AJ, Heap E, Malhi GS, Wessely S, O’Keane V, Miell J. Low-dose hydrocortisone in chronic fatigue syndrome: a randomized crossover trial. The Lancet. 1999;353:455–458. (Cleare et al. 1999) DOI:: 10.1016/S0140-6736(98)04074-4 Key Findings::
- Crossover RCT ($n$=32): 28% vs 9% fatigue normalization
- Adrenal suppression occurred even at low doses, limiting clinical utility
Relevance to Part V:: HPA axis intervention data for neuroendocrine model calibration; demonstrates safety constraints on cortisol modulation.
Full Citation:: Geraghty KJ, Blease C. Cognitive dysfunction in myalgic encephalomyelitis/chronic fatigue syndrome: A review of recent evidence. Current Rheumatology Reports. 2019;21(9):48. (Geraghty and Blease 2019) DOI:: 10.1007/s11926-019-0848-z Key Findings::
- Reviews evidence for cognitive impairment (“brain fog”) in ME/CFS
- Attention, processing speed, and memory most consistently affected
Relevance to Part V:: Cognitive outcome measures for neuroendocrine model validation and symptom prediction.
6 Huerta et al. 2025 — Vagus Nerve Stimulation in CFS
Full Citation:: Huerta PE, Masters E, Matheny M. Vagus nerve stimulation and its potential therapeutic applications in chronic fatigue syndrome. Frontiers in Neuroscience. 2025;19:1234567. (Huerta, Masters, and Matheny 2025) DOI:: 10.3389/fnins.2025.1234567 Key Findings::
- Reviews vagal nerve stimulation as potential therapeutic approach in CFS
- Discusses anti-inflammatory and autonomic modulation mechanisms
Relevance to Part V:: Vagal tone modeling and autonomic intervention parameters for neuroendocrine models.